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GPR82 Receptor Antibodies

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Validation of the GPR82 Receptor in transfected HEK293 cells
GPR82 (non-phospho), G protein-coupled...
The non-phospho-GPR82 receptor antibody is directed against the distal end of the carboxyl-terminal tail of human GPR82. It can be used to detect total GPR82 receptors in Western blots independent of phosphorylation. The GPR82 antibody...
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GPR82 (G Protein-Coupled Receptor 82) is an orphan GPCR with no confirmed endogenous ligand and limited functional characterization. It belongs to the class A family of GPCRs and is conserved across several species, suggesting potential biological relevance. GPR82 is expressed in various tissues, with notable levels in the brain, heart, kidneys, and testis. Some studies have also detected its expression in adipose tissue, indicating a possible role in metabolic regulation. GPR82 is not currently a major focus of drug development, but genetic studies have linked it to metabolic traits, including body mass index and lipid metabolism. While it is not yet considered an established pharmacological target, its potential role in energy balance and metabolic disorders warrants further investigation. For more information on GPR82 pharmacology please refer to the IUPHAR database. For further reading refer to:

Davenport AP, Alexander SP, Sharman JL, Pawson AJ, Benson HE, Monaghan AE, Liew WC, Mpamhanga CP, Bonner TI, Neubig RR, Pin JP, Spedding M, Harmar AJ. International Union of Basic and Clinical Pharmacology. LXXXVIII. G protein-coupled receptor list: recommendations for new pairings with cognate ligands. Pharmacol Rev. 2013 May 17;65(3):967-86. doi: 10.1124/pr.112.007179. PMID: 23686350; PMCID: PMC3698937.

Alexander SP, Battey J, Benson HE, Benya RV, Bonner TI, Davenport AP, Dhanachandra Singh K, Eguchi S, Harmar A, Holliday N, Jensen RT, Karnik S, Kostenis E, Liew WC, Monaghan AE, Mpamhanga C, Neubig R, Pawson AJ, Pin JP, Sharman JL, Spedding M, Spindel E, Stoddart L, Storjohann L, Thomas WG, Tirupula K, Vanderheyden P. Class A Orphans in GtoPdb v.2023.1. IUPHAR/BPS Guide to Pharmacology CITE. 2023; 2023(1). Available from: https://doi.org/10.2218/gtopdb/F16/2023.1.

GPR82 (G Protein-Coupled Receptor 82) is an orphan GPCR with no confirmed endogenous ligand and limited functional characterization. It belongs to the class A family of GPCRs and is conserved... read more »
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GPR82 Receptor Antibodies

GPR82 (G Protein-Coupled Receptor 82) is an orphan GPCR with no confirmed endogenous ligand and limited functional characterization. It belongs to the class A family of GPCRs and is conserved across several species, suggesting potential biological relevance. GPR82 is expressed in various tissues, with notable levels in the brain, heart, kidneys, and testis. Some studies have also detected its expression in adipose tissue, indicating a possible role in metabolic regulation. GPR82 is not currently a major focus of drug development, but genetic studies have linked it to metabolic traits, including body mass index and lipid metabolism. While it is not yet considered an established pharmacological target, its potential role in energy balance and metabolic disorders warrants further investigation. For more information on GPR82 pharmacology please refer to the IUPHAR database. For further reading refer to:

Davenport AP, Alexander SP, Sharman JL, Pawson AJ, Benson HE, Monaghan AE, Liew WC, Mpamhanga CP, Bonner TI, Neubig RR, Pin JP, Spedding M, Harmar AJ. International Union of Basic and Clinical Pharmacology. LXXXVIII. G protein-coupled receptor list: recommendations for new pairings with cognate ligands. Pharmacol Rev. 2013 May 17;65(3):967-86. doi: 10.1124/pr.112.007179. PMID: 23686350; PMCID: PMC3698937.

Alexander SP, Battey J, Benson HE, Benya RV, Bonner TI, Davenport AP, Dhanachandra Singh K, Eguchi S, Harmar A, Holliday N, Jensen RT, Karnik S, Kostenis E, Liew WC, Monaghan AE, Mpamhanga C, Neubig R, Pawson AJ, Pin JP, Sharman JL, Spedding M, Spindel E, Stoddart L, Storjohann L, Thomas WG, Tirupula K, Vanderheyden P. Class A Orphans in GtoPdb v.2023.1. IUPHAR/BPS Guide to Pharmacology CITE. 2023; 2023(1). Available from: https://doi.org/10.2218/gtopdb/F16/2023.1.

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